From the results, it can be concluded that present findings do not support the existence of
association between the 5-HTT gene variant and autism in our subjects, since the short variant
of 5HTT is transmitted but the mutation in 5-HTT may be due to emergence of symptoms of
hyperactivity in patient with gene mutant .The researchers’ findings in autism are preliminary
and require replication. If replicated with patient samples from same and different ethnic and
geographical backgrounds, it is possible that autistic disorder may share common risk at this
locus. Integration of results from DNA sequencing, molecular cytogenetic, and psychiatry
will help us to understand the genetic background of autism in the future.